SELECTED PUBLICATIONS

Primary Research Articles

Black JA, Newcombe J, Trapp BD, Waxman SG.  Sodium channel expression within chronic multiple sclerosis plaques. J Neuropathol Exp Neurol. 2007 Sep;66(9):828-37.

Cummins TR, Sheets PL, Waxman SG.  The roles of sodium channels in nociception: Implications for mechanisms of pain.  Pain. 2007 Oct;131(3):243-57. Epub 2007 Sep 4.

Zhao P, Waxman SG, Hains BC.  Modulation of thalamic nociceptive processing after spinal cord injury through remote activation of thalamic microglia by cysteine cysteine chemokine ligand 21.  J Neurosci. 2007 Aug 15;27(33):8893-902.

Waxman SG.  Nav1.7, its mutations, and the syndromes that they cause. Neurology. 2007 Aug 7; 69(6):505-7.

Black JA, Liu S, Carrithers M, Carrithers LM, Waxman SG.  Exacerbation of experimental autoimmune encephalomyelitis after withdrawal of phenytoin and carbamazepine. Ann Neurol. 2007 Jul; 62(1):21-33.

Hains BC, Waxman SG.  Sodium channel expression and the molecular pathophysiology of pain after SCI. Prog Brain Res. 2007;161:195-203.

Carrithers MD, Dib-Hajj S, Carrithers LM, Tokmoulina G, Pypaert M, Jonas EA, Waxman SG.  Expression of the voltage-gated sodium channel NaV1.5 in the macrophage late endosome regulates endosomal acidification.  J Immunol. 2007 Jun 15;178(12):7822-32.

Patrick Harty T, Waxman SG.  Inactivation properties of sodium channel Nav1.8 maintain action potential amplitude in small DRG neurons in the context of depolarization.  Mol Pain. 2007 May 31;3:12.

Sheets PL, Jackson JO 2nd, Waxman SG, Dib-Hajj SD, Cummins TR.  A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity.  J Physiol. 2007 Jun 15;581(Pt 3):1019-31. Epub 2007 Apr 12.

Neurobiology: a channel sets the gain on pain.  Nature. 2006 Dec 14;444(7121):831-2.

Na(V)1.7 mutant A863P in erythromelalgia: effects of altered activation and steady-state inactivation on excitability of nociceptive dorsal root ganglion neurons.
J Neurosci. 2006 Nov 29;26(48):12566-75.

Choi JS, Dib-Hajj SD, Waxman SG.  Differential slow inactivation and use-dependent inhibition of Nav1.8 channels contribute to distinct firing properties in IB4+ and IB4- DRG neurons. J Neurophysiol. 2007 Feb;97(2):1258-65. Epub 2006 Nov 15.

Craner MJ. Damarjian TG. Liu S. Hains BC. Lo AC. Black JA. Newcombe J. Cuzner ML. Waxman SG. Sodium channels contribute to microglia/macrophage activation and function in EAE and MS. GLIA. 49(2):220-9, 2005 Jan 15.

Waxman SG. Craner MJ. Black JA. Na+ channel expression along axons in multiple sclerosis and its models. Trends in Pharmacological Sciences. 25(11):584-91, 2004 Nov.

Wittmack EK. Rush AM. Craner MJ. Goldfarb M. Waxman SG. Dib-Hajj SD. Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons. Journal of Neuroscience. 24(30):6765-75, 2004 Jul 28.

Craner MJ. Hains BC. Lo AC. Black JA. Waxman SG. Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE. Brain. 127:294-303, 2004 Feb.

Radtke C. Akiyama Y. Brokaw J. Lankford KL. Wewetzer K. Fodor WL. Kocsis JD. Remyelination of the nonhuman primate spinal cord by transplantation of H-transferase transgenic adult pig olfactory ensheathing cells. FASEB Journal. 18(2):335-7, 2004 Feb.

Lo AC. Saab CY. Black JA. Waxman SG. Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo. Journal of Neurophysiology. 90(5):3566-71, 2003 Nov.

Klein JP. Tendi EA. Dib-Hajj SD. Fields RD. Waxman SG. Patterned electrical activity modulates sodium channel expression in sensory neurons. Journal of Neuroscience Research. 74(2):192-8, 2003 Oct 15.

Saab CY. Cummins TR. Waxman SG. GTP gamma S increases Nav1.8 current in small-diameter dorsal root ganglia neurons. Experimental Brain Research. 152(4):415-9, 2003 Oct.

Baker MD. Chandra SY. Ding Y. Waxman SG. Wood JN. GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones. Journal of Physiology. 548(Pt 2):373-82, 2003 Apr 15.

Craner MJ. Kataoka Y. Lo AC. Black JA. Baker D. Waxman SG. Temporal course of upregulation of Na(v)1.8 in Purkinje neurons parallels the progression of clinical deficit in experimental allergic encephalomyelitis. Journal of Neuropathology & Experimental Neurology. 62(9):968-75, 2003 Sep.

Hains BC. Klein JP. Saab CY. Craner MJ. Black JA. Waxman SG. Upregulation of sodium channel Nav1.3 and functional involvement in neuronal hyperexcitability associated with central neuropathic pain after spinal cord injury. Journal of Neuroscience. 23(26):8881-92, 2003 Oct 1.

Herzog RI. Liu C. Waxman SG. Cummins TR. Calmodulin binds to the C terminus of sodium channels Nav1.4 and Nav1.6 and differentially modulates their functional properties. Journal of Neuroscience. 23(23):8261-70, 2003 Sep 10.

Inoue M, Honmou 0, Oka S, Houkin K, Hashi K and Kocsis JD. Comparative analysis of remyelinating potential of focal and intravenous administration of autologous bone marrow cells into the rat demyelinated spinal cord. GLIA. 44: 111-118, 2003.

Klein JP. Waxman SG. The brain in diabetes: molecular changes in neurons and their implications for end-organ damage. Lancet Neurology. 2(9):548-54, 2003 Sep.

Tokuno HA. Kocsis JD. Waxman SG. Noninactivating, tetrodotoxin-sensitive Na+ conductance in peripheral axons. Muscle & Nerve. 28(2):212-7, 2003 Aug.

Craner MJ. Lo AC. Black JA. Waxman SG. Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination. Brain. 126:1552-61, 2003 Jul.

Hains BC. Black JA. Waxman SG. Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury. Journal of Comparative Neurology. 462(3):328-41, 2003 Jun.

Liu, C., Dib-Hajj, S., Renganathan, M., Cummins, T., Waxman, S. Modulation of cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B. J. Biol. Chem. 278:1029-1036, 2003.

Renganathan, M., Gelderblom, M., Black, J., and Waxman, S. Expression of Nav 1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells. Brain Res. 959, 235-242, 2003.

Craner, M., Klein, J., Renganathan, M., Black, J., and Waxman, S. Changes of sodium channel expression in experimental painful diabetic neuropathy. Annals of Neurology. 52:786-92, 2002.

Waxman, S.G., Sodium channels as molecular targets in multiple sclerosis. Journal of Rehabilitation Research and Development 39: 233-242, 2002.

Fang, X., Djouhri, L., Black, J. A., Dib-Hajj, S. D., Waxman, S. G., Lawson, S. N. The presence and role of the TTX resistant sodium channel Nav1.9 (NaN) in nociceptive primary afferent neurons. J. Neurosci. 22: 7425-7433, 2002.

Lo, A., Black, J., and Waxman, S. Neuroprotection of axons with phenytoin in EAE. NeuroReport 13, 1909-12, 2002.

Herzog, R.I., Cummins, T.R., and Waxman, S.G. (2001) Persistent TTX-resistant sodium current affects resting potential and excitability in simulated spinal sensory neuron. Journal of Neurophysiology, 86(3): 1351-1364.

Renganathan, M., Cummins, T.R., and Waxman, S.G. (2001) The contribution of Nav1.8 sodium channels to action potential electrogenesis in DRG neurons. Journal of Neurophysiology, 86: 629-640.

Liu, C., Dib-Hajj, S.D., and Waxman, S.G. (2001) Fibroblast growth factor homologous factor 1B binds to the c-terminus of the tetrodotoxin-resistant sodium channel rNav1.9a (NaN). Journal of Biological Chemistry, 276(22):18925-18933.

Sasaki, M., Honmou, O., Akiyama, Yukinori., Uede, T., Hashi, K., and Kocsis, J.D. (2001) Remyelination of spinal cord by intravenous delivery of bone marrow cells. GLIA, 35: 26-34.

Kohama I. Lankford KL. Preiningerova J. White FA. Vollmer TL. Kocsis JD. (2001) Transplantation of cyropreserved adult human Schwann cells enhances axonal conduction in demyelinated spinal cord. Journal of Neuroscience, 21:944-950.

Renganathan, M., Cummins, T. R., Hormuzdiar, W. N., Black, J. A., Waxman, S. G.  (2000) Nitric oxide is an autocrine regulator of Na+ currents in axotomized C-type DRG neurons. Journal of Neurophysiology, 83(4):2431-2442.

Baccei, M.L., and Kocsis, J.D.  (2000) Voltage-gated calcium currents in axotomized adult rat cutaneous afferent neurons.  Journal of Neurophysiology, 83:2227-2238.

Kohama, I., Ishikawa, K., and Kocsis, J.D.  (2000) Synaptic reorganization in the substantia gelatinosa after peripheral nerve neuroma formation: aberrant innervation of lamina II neurons by Ab afferents.  Journal of Neuroscience. 20, 4.

Imaizumi, T., Lankford, K.L., Kocsis, J.D.  (2000) Transplantation of olfactory ensheathing or Schwann cells restores rapid and secure conduction across the transected spinal cord. Brain Research.

Fjell, J., Hjelmstrom, P., Hormuzdiar, W., Milenkovic, M., Aglieco, F., Tyrrell, L., Dib-Hajj, S., Waxman,S.G., Black, J.A.  (2000) Localization of the tetrodotoxin-resistant sodium channel NaN in nociceptors.  NeuroReport, 11: 199-203.

Black, J.A., Fjell, J., Dib-Hajj, S., Duncan, I.D., O’Connor, L.T., Fried, K., Gladwell, Z., Tate, S., Waxman, S.G.  (1999) Abnormal expression of SNS/PN3 sodium channel in cerebellar Purkinje cells following loss of myelin in the taiep rat. NeuroReport, 10:913-918.

Dib-Hajj, S.D., Tyrrell, L., Escayg, Andrew., Wood, P.M., Meisler, M.H., Waxman, S.G.  (1999) Coding sequence, genomic organization and chromosomal localization of the mouse gene Scn11a encoding the sodium channel NaN. Genomics, 59, 309-318.

Fjell, J., Cummins, T.R., Dib-Hajj, S.D., Fried, K., Black, J.A., and Waxman, S.G.  (1999) Differential role of GDNF and NGF in the maintenance of two TTX-resistant sodium channels in adult DRG neurons.  Molecular Brain Research, 67: 267-282.

Tanaka, M., Cummins, T.R., Ishikawa, K., Black, J.A., Ibata, Y., Waxman, S.G.  (1999) Molecular and functional remodeling of electrogenic membrane of hypothalamic neurons in response to changes in their input. Proceedings of National Academy of Science, 96:1088-1093.

Black, J.A., Cummins, T.R., Plumpton, C., Chen, Y.H., Hormuzdiar, W., Clare, J.J., and Waxman, S.G.  (1999) Upregulation of a silent sodium channel after peripheral, but not central, nerve injury in DRG neurons.  Journal of Neurophysiology, 82: 2776-2785.

Everill, B., Kocsis, J.D.  (1999) Reduction in potassium currents in identified cutaneous afferent DRG neurons after axotomy. Journal of Neurophysiology, 82: 700-708.

Dib-Hajj, S.D., Fjell, J., Cummins, T.R., Zheng, Z., Fried, K., LaMotte, R., Black, J.A., Waxman, S.G.  (1999) Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain. Pain, 83: 591-600.

Cummins, T.R., Dib-Hajj, S.D., Black, J.A., Akopian, A.N., Wood, J.N., Waxman, S.G.  (1999) A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons. Journal of Neuroscience, 19: RC 43 (1-6).

Dib-Hajj, S.D., Tyrrell, L., Cummins, T.R., Black, J.A., Wood, P.M., Waxman, S.G.  (1999) Two tetrodotoxin-resistant sodium channels in human dorsal root ganglion neurons. FEBS Letters, 462: 117-120.

Dib-Hajj, S.D., Tyrrell, L., Black, J.A., Waxman, S.G.  (1998a) NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy. Proceedings of National Academy of Science, 95: 8963-8969.

Lankford, K.L., Waxman, S.G. and Kocsis, J.D.  (1998) Mechanisms of enhancement of neurite regeneration: rat DRG neurite arborization in vitro after a conditioning sciatic nerve lesion in vivo. Journal of Comparative Neurology, 391:11-29.

Imaizumi, T., Kocsis, J.D., and Waxman, S.G.  (1998) Resistance to anoxic injury in rat spinal cord following demyelination. Brain Research, 779:292-296.

Black, J. A., Dib-Hajj, S., Cohen, S., Hinson, A. W., Waxman, S. G.  (1998) Glial cells have heart: rH1 Na+ channel mRNA and protein in spinal cord astrocytes. Glia, 23:200-208.

Cummins, T.R., Howe, J.R., Waxman, S.G.  (1998) Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel. Journal of Neuroscience, 18: 9607-9619.

Dib-Hajj, S.D., Black, J.A., Cummins, T.R., Kenney, A.M., Kocsis, J.D., Waxman, S.G.  (1998) Rescue of a-SNS sodium channel expression in small dorsal root ganglion neurons after axotomy by nerve growth factor in vivo. Journal of Neurophysiology, 79:2668-2676.

Everill, B., Rizzo, M.A., Kocsis, J.D.  (1998) Morphologically identified cutaneous afferent DRG neurons express three different potassium currents in varying proportions. Journal of Neurophysiology, 79: 1814-1824.

Yee J, Agulian S and Kocsis JD.  (1998)  Vigabatrin enhances promoted release of GABA in neonatal rat optic nerve.  Epilepsy Research, 29:195-200.

Imaizumi, T., Lankford, K.L., Waxman, S.G., Greer, C.A., Kocsis, J.D.  (1998) Transplanted olfactory ensheathing cells remyelinate and enhance axonal conduction in the demyelinated dorsal columns of the rat spinal cord.  Journal of Neuroscience, 18(16): 6176-6185.

Robert, A., Black, J.A., Waxman, S.G.  (1998) Endogenous NMDA receptor activation regulates glutamate release in cultured spinal neurons.  Journal of Neurophysiology, 80:196-208.

Sashihara, S., Waxman, S.G., and Greer, C.A.  (1997) Down-regulation of Na+ channel mRNA following sensory deprivation of tufted cells in the neonatal rat olfactory bulb.  NeuroReport 8:1289-1293.

Oyelese, A.A., Rizzo M.A., Waxman, S.G. and Kocsis, J.D.  (1997) Differential effects of NGF and BDNF on axotomy-induced changes in GABAA  receptor-mediated conductance and sodium currents in cutaneous afferent neurons. Journal of Neurophysiology, 78:31-42.

Hinson, A.W., Gu, X.Q., Dib-Hajj, S.D., Black, J.A. and Waxman, S.G.  (1997) Schwann cells modulate sodium channel expression in spinal sensory neurons in vitro. Glia, 21:339-349.

Imaizumi, T., Kocsis, J.D., and Waxman, S.G.  (1997) Anoxic injury in the rat spinal cord:  pharmacological evidence for multiple steps in Ca2+-dependent injury of the dorsal columns.  Journal of Neurotrauma, 14: 293-312.

Black, J.A., Langworthy, K., Hinson, A.W., Dib-Hajj, S.D., and Waxman, S.G.  (1997) NGF has opposing effects on Na+ channel III and SNS gene expression in spinal sensory neurons. NeuroReport,  8: 2331-2335.

Cummins, T.R. and Waxman, S.G.  (1997) Down-regulation of tetrodotoxin-resistant sodium currents and up-regulation of a rapidly repriming tetrodotoxin-sensitive sodium current in small spinal sensory neurons following nerve injury.  Journal of Neuroscience, 17:3503-3514.

Dib-Hajj, S., D., Ishikawa, I., Cummins, T.R., and Waxman, S.G.  (1997) Insertion of a SNS-specific tetrapeptide in the S3-S4 linker of D4 accelerates recovery from inactivation of skeletal muscle voltage-gated Na channel µ1 in HEK293 cells. FEBS Letters, 416:11-14.

Honmou, O., Felts, P.A., Waxman, S.G., and Kocsis, J.D.  (1996) Restoration of normal conduction properties in demyelinated spinal cord axons in the adult rat by transplantation of exogenous Schwann cells.  Journal of Neuroscience, 16:3199-3208.

Rizzo, M.A., Kocsis, J.D. and Waxman, S.G.  (1996) Mechanisms of paraesthesiae, dysaesthesiae, and hyperaesthesiae:  Role of Na channel heterogeneity. Eur. Neurol., 36:3-12.

Waxman, S.G., and Rizzo, M.A.  (1996) The whiplash (hyperextension-flexion) syndrome--A disorder of dorsal root ganglion neurons? Journal of Neurotrauma, 13:735-740.

Dib-Hajj, S.D., Black, J.A., Felts, P., Waxman, S.G.  (1996) Down-regulation of transcripts for sodium channel a-SNS in spinal sensory neurons following axotomy. Proceedings of National Academy of Science, 93: 14950-14954.

Roy, M.L., Saal, D., Perney, T., Sontheimer, H., Waxman, S.G., and Kaczmarek, L.K.  (1996) Manipulation of the delayed rectifier Kv1.5 potassium channel in glial cells by antisense oligodeoxynucleotides.  Glia, 18:177-184.

Honmou, O., Felts, P.A., Waxman, S.G., Kocsis, J.D.  (1996) Restoration of normal conduction properties in demyelinated spinal cord axons in the adult rat by transplantation of exogenous Schwann cells.  Journal of Neuroscience, 16: 3199-3208.

Black, J.A., Dib-Hajj, S.D., McNabola, K., Jeste, S., Rizzo, M.A., Kocsis, J.D., Waxman, S.G.  (1996) Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs.  Molecular Brain Research, 43: 117-131.

Fern, R., Waxman, S.G., and Ransom, B.R.  (1995) Endogenous GABA attenuates CNS white matter dysfunction following anoxia.  Journal of Neuroscience, 15:699-708.

Azizi, S.A., Fayad, P.B., Fulbright, R., Giroux, M.l. and Waxman, S.G.  (1995) Clivus and cervical spinal osteomyelitis with epidural abscess presenting with multiple cranial neuropathies.  Clinical Neurology and Neurosurgery, 97:239-244.

Felts, P.A., Black, J.A., Oh, Y., and Waxman, S.G.  (1995) Expression of Sodium Channel a-and ß-subunits in the nervous system of the myelin deficient rat.  Journal of Neurocytololgy., 24:654-666.

Waxman, S.G., Kocsis, J.K., Black, J.A.  (1994) Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotonomy.  Journal of Neurophysiology, 72: 466-471.

Waxman, S.G., Utzschneider, D.A., and Kocsis, J.D.  (1994) Enhancement of action potential conduction following demyelination:  Experimental approaches to restoration of function in multiple sclerosis and spinal cord injury.  Progress in Brain Research, 100: 233-244.

Utzschneider, D.A., Archer, D.R., Kocsis, J.D., Waxman, S.G. and Duncan,   (1994) Transplantation of glial cells enhances action potential conduction of amyelinated spinal cord axons in the myelin-deficient rat. Proceedings of National Academy of Science, 91:53-57.

Black, J.A., Yokoyama, S., Waxman, S.G., Oh, Y., Zur, K.B., Sontheimer, H., Higashida, H., and Ransom, B.R.  (1994) Sodium channel mRNAs in cultured spinal cord astrocytes:  in situ hybridization in identified cell types.  Molecular Brain Research, 23:235-245.

Oh, Y., and Waxman, S.G.  (1994) The ß1 subunit mRNA of the rat brain Na+ channel is expressed in glial cells.  Proceedings of National Academy of Science, 91:9985-9989.

Thio, C.L., Waxman, S.G., and Sontheimer, H.  (1993) Ion channels in spinal cord astrocytes in vitro:  III. Modulation of channel expression by co-culture with neurons and neuron-conditioned medium. Journal of Neurophysiology, 69:819-831.

Waxman, S.G., Black, J.A., Ransom, B.R., and Stys, P.K.  (1993) Protection of the axonal cytoskeleton in anoxic optic nerve by decreased extracellular calcium.  Brain Research, 614:137-145.

Stys, P.K., Waxman, S.G. and Ransom, B.R.  (1992) Ionic mechanisms of anoxic injury in mammalian CNS white matter: Role of Na+ channels and Na+-Ca2+ exchanger.  Journal of. Neuroscience, 12:430-439.

Waxman, S.G., Black, J.A., Stys, P.K. and Ransom, B.R.  (1992) Ultrastructural concomitants of anoxic injury and early post-anoxic recovery in rat optic nerve. Brain Research, 574: 105-119.

Stys, P.K., Ransom, B.R. and Waxman, S.G.  (1992) Tertiary and quaternary local anesthetics protect CNS white matter from anoxic injury at concentrations that do not block excitability. Journal of Neurophysiology, 67:236-240.

Sontheimer, H., Black, J.A., Ransom, B.R., and Waxman, S. G.  (1992) Ion channels in spinal cord astrocytes in vitro:  I.  Transient expression of high levels of Na+ and K+ channels. Journal of Neurophysiology, 68:985-999.

Sontheimer, H., and Waxman, S.G.  (1992) Ion channels in spinal cord astrocytes in vitro:  II.  Biophysical and pharmacological analysis of two Na+ current types.  Journal of Neurophysiology, 68:1000-1011.

Kocsis, J.D., Eng, D.L., Gordon, T.R. and Waxman, S. G.  (1987)Functional differences between 4-aminopyridine and tetraethylammonium-sensitive potassium channels in myelinated axons.  Neuroscience Letters, 75:193-198.

Targ, E.F. and Kocsis, J.D.  (1985) 4-aminopyridine leads to restoration of conduction in demyelinated rat sciatic nerve.  Brain Research, 328:358-361.

Review Articles, Book Chapters and Editorials

Black, J.A., Cummins, T.R., Dib-Hajj S.D., and Waxman, S.G. (2002) Sodium channels and the molecular basis for pain. In: Mechanisms and Mediators of Neuropathic Pain, Malmberg, A., and Chaplan, S.R. pp 23-50.

Waxman, S.G. (2001) Transcriptional Channelopathies: An emerging class of disorders. Nature Neuroscience, 2(9): 652-659.

Waxman, S. G., and Kocsis, J. D.  (2000) Experimental approaches to restoration of function of ascending and descending axons in spinal cord injury. In: The Neurobiology of Spinal Cord Injury, R.G. Kalb and S. M. Strittmatter, Eds., Humana Press, pp. 215-240.

Kocsis, J.D.  (1999) Restoration of function by glial cell transplantation into demyelinated spinal cord. Journal of Neurotrauma, 16(9): 695-703.

Waxman S.G., Dib-Hajj S., Cummins T.R., Black J.A.  (1999) Sodium channels and Pain. Proceedings of National Academy of Science, 96: 7635-7639.

Kocsis, J.D., and Waxman, S.G.  (1998) Transplantation of myelin-forming cells into the spinal cord: restoration of normal conduction in previously demyelinated axons.  In: Glial Cells and Behavior, P. Laming (Ed.), Cambridge University Press. pp 181-198.

Waxman, S.G.  (1998) Demyelinating diseases:  New pathological insights, new therapeutic targets. New England Journal of Medicine, 338:323-325.

Waxman, S.G.  (1997) Axon-Glia interactions: Building a smart nerve fiber. Current Biology, 7(7): R406-R410.

Waxman, S.G.  (1997) Molecular remodelling of neurons in multiple sclerosis:  What we know, and what we must ask about brain plasticity in demyelinating diseases. In: Brain Plasticity,  H-J Freund, B.A. Sabel and O.W. White (eds.), Raven Press, New York, pp 109-121.

Waxman, S. G.  (1992) Demyelination in spinal cord injury and multiple sclerosis: What can we do to enhance functional recovery? Journal of Neurotrauma, 9:S105-S117.

Waxman, S. G., Ransom, B. R. and Stys, P. K.  (1991) Non-synaptic mechanisms of calcium mediated injury in CNS white matter. Trends in Neuroscience, 14:461-468.

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